P Palthera
Signalling Peptides

Dermorphin

Dermorphin (Phyllomedusa)

Dermorphin is a seven- opioid peptide isolated from the skin of South American Phyllomedusa frogs in the 1980s. It is a highly selective μ-opioid and is approximately 30–40 times more potent than morphine in animal models. Dermorphin and its are research compounds; the parent compound has documented illicit use in horse racing and is treated as a controlled substance for veterinary and anti-doping purposes.

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Signalling Peptides
Classification
Heptapeptide μ-opioid receptor agonist isolated from Phyllomedusa frog skin · Controlled substance / WADA prohibited
Research stage
Pharmacology and structure-activity literature from the 1980s onwards; analogue development for analgesia research
Sequence
Tyr-D-Ala-Phe-Gly-Tyr-Pro-Ser-NH2
Molecular weight
802.9 Da

Snapshot

Key takeaways

A three-bullet snapshot before reading the full dossier.

  1. 01

    Highly selective μ-opioid 30–40× more potent than morphine in animal models.

  2. 02

    Originally isolated from Phyllomedusa frog skin; multiple (e.g. DALDA, DAMGO) extend its pharmacology.

  3. 03

    Controlled substance / WADA prohibited substance — not for human use outside authorised research.

Dossier overview

4

research areas

3

references

3

handling notes

01

Mechanism of action

Dermorphin is a potent at the μ-opioid (MOR) with substantially higher affinity than morphine. Its D-Ala at position 2 confers proteolytic and is essential for activity. research has focused on peripherally restricted variants to reduce centrally-mediated side effects.

02

Research applications

  • μ-opioid pharmacology
  • Opioid and structure-activity research
  • Peripheral analgesia research (DALDA and related variants)
  • Anti-doping detection methodology

Evidence at a glance

What's behind this profile

3 citations · 1988–2024

Animal
1

Studies in rodents or other animal models.

In vitro
1

Cell, tissue, or biochemical assays outside a living organism.

Review
1

Narrative or systematic reviews; no primary data.

Publication years

  1. 88
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19882024

Counts are derived from the cited studies below. A study covering both in vivo and in vitro work is counted by its primary model. Sample size is reported in 0 of 3 citations. Findings remain model-specific and are not extrapolated to therapeutic use.

03

Study references

Each profile cites a minimum of two peer-reviewed sources, with model type and reported sample size where the source provides it. Findings are model-specific and must not be extrapolated to therapeutic use.

Opioid peptide-derived analgesics

2005

Schiller PW et al. · AAPS Journal

Model
Narrative review of opioid peptide analgesic research
Sample
N/A (review)

Reviewed dermorphin-derived analgesics (incl. DALDA and mixed μ/δ modulators) framed for reduced tolerance and dependence profiles in models.

PMID 16353933 DOI 10.1208/aapsj070356

Structural requirements for dermorphin opioid receptor binding

1988

Amiche M et al. · International Journal of Peptide and Protein Research

Model
In vitro receptor binding / structure-activity study
Sample
N/A (biochemical assay)

Established that the D-Ala2 configuration and C-terminal are critical structural features for dermorphin's high-affinity μ-opioid binding.

PMID 2906053 DOI 10.1111/j.1399-3011.1988.tb00922.x

Peripheral μ-opioid receptor activation by dermorphin alleviates behavioural and neurobiological aberrations in chemotherapy-induced neuropathic pain

2024

Gadepalli A et al. · Neurotherapeutics

Model
In vivo — rat model of paclitaxel-induced neuropathic pain (DALDA peripheral μ-agonist)
Sample
Not reported in abstract

Reported that peripheral μ-opioid activation by a dermorphin-derived (DALDA) was associated with reduced behavioural and neurobiological markers of paclitaxel-induced neuropathic pain in rats.

PMID 38241153 DOI 10.1016/j.neurot.2023.10.012

Evidence caveats

  • Dermorphin and its analogues are controlled substances in many jurisdictions and WADA-prohibited. Research access is restricted; clinical / unsupervised use is not endorsed.
  • Published primary evidence on the parent dermorphin compound is concentrated in 1980s–1990s pharmacology; modern research focuses on analogues such as DALDA.

04

Storage and handling

Store under controlled laboratory conditions, in secured controlled-substance inventory where applicable, with batch and chain-of-custody details recorded.

  • Dermorphin is treated as a controlled substance in many jurisdictions due to its opioid potency.
  • Research-only inventory under strict access controls; subject to local controlled-substance regulations.
  • Verify supplier provenance and document for every transfer.